Monday, March 9, 2009

Pediatric Nursing: skin disorders/burns


Skin – Immune Disorders


Key Function of Skin

Protection – shield from internal injury.


Immunity – contains cells that ingest bacteria and other substances.


Thermoregulation – heat regulation through sweating, shivering, and
subcutaneous insulation


Communication / sensation / regeneration



Developmental Variances

Sweat glands function by the time the child is 3-years-old.


The visco-elastic property of the dermis becomes completely functional at
about 2 years.


The neonate’s dermis is thin and very hydrated, thus is at greater risk for
fluid loss and serves as an ineffective barrier.



Diagnostic Tests


Cultures


Scraping


Skin biopsy


Skin testing


Woods lamp



Neonatal skin lesions





Vascular birth marks: hemangioma


Port wine stain


Abnormal pigmentation: Mongolian spots


Neonatal acne: small red papules and pustules appear on face trunk.


Milia: white or yellow, 1-2mm papules appearing on cheeks, nose, chin, and
forehead



Inflammatory Skin Disorders





Diaper dermatitis


Contact dermatitis


Atopic dermatitis or eczema



Diaper Dermatitis


Diaper Dermatitis





Identify causative agent


Cleanse with mild cleaner


Apply barrier


Expose to air


Teach hazards of baby powder



Cradle Cap





Rash that occurs on the scalp.


It may cause scaling and redness of the scalp.


It may progress to other areas.



Treatment





If confined to the scalp


Wash area with mild baby shampoo and brush with a soft brush to help remove
the scales.


Do not apply baby oil or mineral oil to the area - this will only allow for
more build up of the scales.



Cradle Cap


Baby Care


Atopic dermatitis or Eczema





Chronic, relapsing inflammation of the dermis and epidermis characterized
by itching, edema, papules, erythema, excoriation, serous discharge and
crusting.


Patients have a heightened reaction to a variety of allergens.



Dermatitis


Dermatitis


Assessment





History of asthma, allergic rhinitis


Lesions generally occur in creases.


Management:





Control the itching: OTC Benadryl


Reduce inflammation: topical corticosteroids


Hydrate the skin


Elidel Cream


Preventing infection






Acne Vulgaris





A chronic, inflammatory process of the pilosebaceous follicles.


Occurrence; 85% of teenager aged 15 to 17 years.


More common in females than males.



Acne





Over activity of oil glands at the base of hair follicles


Hormone activity


Skin cell "plug" pores causing white heads and blackheads.


No "cure"



Acne


Management of Acne





Topical medications





OTC preparations


Prescription - Topical retinoid preparations


Prescription - Topical antibiotics




Systemic medication





Antibiotics


Hormonal therapy – birth control pills


Accutane * use with extreme caution when all else fails





Pediculosis





Head lice infestation ranges from 1% to 40% in children.


Most common in ages 5 to 12.


Less common in African American due to the shape of the hair shaft.


Transmission by direct contact with infected person, clothing, grooming
articles, bedding, or carpeting.



Pediculosis





Head lice


Pubic lice


Body lice



Signs and Symptoms





Symptoms: itching, whitish colored eggs at shaft of hair, redness at site
of itching.



Nits


Interventions






Anti-lice shampoo


Removal of nits


Washing bedding, towels, anything child’s head may have
come in contact with in hot soapy water.


Vacuum all floors and rugs


Do not need to fumigate the house


Child can return to school after 1 day of treatment






Scabies





A contagious skin condition caused by the human skin mite.


Tiny, eight-legged creature burrows within the skin and penetrate the
epidermis and lays eggs


Allergic reaction occurs


Severe itching



Assessment





Pruritus especially profound at night or nap time.


Lesions may be generalized but tend to distribute on the palms, soles and
axillae


In older children: finger webs, body creases, beltline and genitalia



Management





Permethrin cream is drug of choice


Massage into all skin surfaces – neck to soles of feet - leave on for 8 to
14 hours.


Clothing bedding and other contact items need to be washed in hot soapy
water.


Vacuum upholstered furniture - rugs



Scabies


Scabies


Impetigo





The most common skin infection in children.


Causative agent is carried in the nasal area.


Bacteria invade the superficial skin.



Causative agent





Group A beta-hemolytic streptococcal (GABHS)


Staph aureus



Impetigo


Spread





Highly contagious skin infection.


Most common among children.


Spread through physical contact.


Clothes, bedding, towels and other objects.



Interventions


Outcomes





Self-limiting


No scarring or pox marks post infection.


Super-infection especially in the neonate.



Impetigo / cellulitis


Cellulitis





A full-thickness skin infection involving dermis and underlying connective
tissue.


Any part of the body can be affected.


Cellulitis around the eyes is usually an extension of a sinus infection or
otitis media.



Assessment





History and physical exam


WBC count


Blood culture


Culturing organism from lesion aspiration.


CT scan with peri-orbital cellulitis






Clinical Manifestations





Characteristic reddened or lilac-colored, swollen skin that pits when
pressed with finger.


Borders are indistinct.


Warm to touch.


Superficial blistering.



Cellulitis


Cellulitis


Interventions





Hospitalization if large area involved or facial cellulitis.


IV antibiotics.


Tylenol for pain management.


Warm moist packs to area if ordered.


Assess for spread


If peri-orbital test for ocular movement and vision acuity



Poison Oak, Ivy and Sumac





Three potent antigens that characteristically produce an intense
dermatologic inflammatory reaction when contact is made between the skin and
the allergens contained in the plant.



Poison Ivy


Poison Oak


Interventions





Prevention:


Wear long pants when hiking or playing in the brush.


Wash with soap and water to remove sticky sap.


Cleanse under finger nails.


Sap on fur, clothing or shoes can last up to 1 week if not cleansed
properly.


Topical cortisone to lesions.



Systemic Response


Burns in Children





Young children who have been severely burned have a higher mortality rate
than adults.


Shorter exposure to chemicals or temperature can injure child sooner.


Increased risk for for fluid and heat loss due to larger body surface area.



Burns in Children





Burns involving more that 10% of TBSA require fluid resuscitation.


Infants and children are at increased risk for protein and calorie
deficiency due to decreased muscle mass and poor eating habits.


Scarring in more severe.



Burns in Children





Immature immune system can lead to increased risk of infection.


Delay in growth may follow extensive burns.






Management of Burns





Ascertain adequacy of airway, oxygen, intubation


Large bore needle to deliver sufficient fluids at a rapid rate.


Remove clothing and jewelry and examine.



Alert





The most common cause of unconsciousness in the flame burn patient is
hypoxia due to smoke inhalation.


Look for ash and soot around nares.



Management of Burns





Admission weight.


Nasogastric tube to maintain gastric decompression.


Foley catheter for urine specimen and monitor output.


Evaluate burn area and determine the extent and depth of injury.



Flame Burn


Management





NG tube in place.


Catheter for fluid replacement.


Ambulation to prevent problems


associated with immobilization



Percentage of Areas Affected


Depth of Burns


First Degree Burn





Involves only the epidermis and part of the underlying skin layers.


Area is hot, red, and painful, but without swelling or blistering.


Sunburn is usually a first-degree burn.



Second Degree Burn





Involves the epidermis and part of the underlying skin layers.


Pain is severe.


Area is pink or red or mottled.


Area is moist and seeping, swollen, with blisters.



Third Degree or Full-thickness





Involves injury to all layers of skin.


Destroys the nerve and blood vessels


No pain at first


Area may be white, yellow, black or cherry red.


Skin may appear dry and leathery.



Wound Management


Wound Management


Wound Management





Hydrotherapy is used to cleanse the wound. Gauze pads are used


To debride the wound by removing exudates and previous applied


Medication.



Skin Grafts


Compartment Syndrome


Burn Wound Covering


Therapy to Prevent Complications


Burns


Keep Kids Safe


Infants Immune System





No active immune response at birth


Passive immunity from mother


Potential for immune response is present / active response is lacking



Immune Response





IgG is received from mother trans-placental and in breast milk


6 to 9 months infants start to produce IgG


Immune system starts to assume defensive role


Active immunity begins after exposure to antigens



Neonatal Sepsis





Can be caused by bacterial, fugal, parasitic or viral pathogens.


Etiology: complex interaction of maternal-fetal colonization,
transplacental immunity and physical and cellular defenses of the fetus and
mother.



Sepsis





Laboratory confirmed blood stream infection






Neonatal sepsis





Mortality rate 50%


1 to 8 cases per 1000 live births


Meningitis occurs in 1/3



Major Risk Factors





Maternal prolonged rupture of membranes > 24 hours


Intra-partum maternal fever > 38C


Prematurity


Sustained fetal tachycardia > 160



Minor Risk Factors





Twin gestation


Premature infant


Low APGAR


Maternal Group B Streptococcus


Foul lochia



Etiology





Group B beta-hemolytic Streptococcus


Escherichia coli


Haemophilus Influenza



Diagnostic Tests





C-Reactive Protein * earliest indicator of infectious / inflammatory
process


CBC with differential



WBC



Blood Culture – rule out blood borne bacteria – sepsis (take 3 days for
final culture results)


Lumbar Puncture – rule out meningitis


Urine Culture – rule out UTI



Clinical Manifestations





Respiratory distress





Tachypnea / apnea / hypoxia



Temperature instability





> 99.6 (37 C) or < 97 (36 C)



Gastrointestinal symptoms





Vomiting, diarrhea, poor feeding



Decreased activity: lethargic / not eating




Blood Test



C-Reactive Protein





Protein appears within 6 hours or exposure


Blood culture to identify causative agent




Medical Management





Ampicillin


Gentamicin


Cefotaxime


Acyclovir: herpes



Nursing Interventions





Administer IV antibiotics


Monitor therapeutic levels


Monitor VS, temperature, O2 saturation


Activity level


Sucking


Infant parent bonding



Outcomes





Newborn will achieve normalization of body function


Parents will participate in care


Newborn will demonstrate no signs of CV, neurological or respiratory
compromise


Newborn will experience no hearing loss as a result of antibiotic therapy



SCIDS





Severe Combined Immunodeficiency Disease


Hereditary disease


Absence of both humoral and cell mediated immunity



Clinical Manifestations





Susceptibility to infection


Frequent infection


Failure of infection to respond to antibiotic treatment



Treatment





Manage infection


Bone marrow transplant



Acquired Immunodeficiency Syndrome / AIDS



Human immunodeficiency virus type 1


is a retro virus that attacks the immune


system by destroying T lymphocytes.



AIDS





T lymphocytes are critical to fighting infection and developing immunity.


HIV renders the immune system useless and the child is unable to fight
infection.



Killer T-cells


Blood Testing in Infants





Babies born to HIV-positive mothers initially test positive for HIV
antibodies.


Only 13 to 39% of these infants are actually infected.


Infants who are not infected with HIV may remain positive until they are
about 18- months-old.



Treating Infants in Utero





Routinely offer HIV testing to all pregnant women.


Administration of zidovudine (AZT) can decrease the likelihood of perinatal
transmission from 25% to 8%.




Modes of Transmission





Three chief modes of transmission:





Sexual contact (both homosexual and heterosexual).


Exposure to needles or other sharp instruments contaminated with blood or
bloody body fluids.


Mother-to-infant transmission before or around the time of birth.






Symptoms in Children





An infant who is HIV positive will generally exhibit symptoms between 9
months to 3 years.


Failure to thrive


Pneumonia, chronic diarrhea, opportunistic infections


Encephalopathy: leading to developmental delay, or loss of previously
obtained milestones.



Interdisciplinary Interventions





Maternal treatment during pregnancy.


Newborn receives zidovudine for 6 weeks after birth.


Prophylaxis with Septra or Bactrim when CD4 level starts to drop.



Interventions





Age-appropriate immunizations except those containing live attenuated
viruses. Can be given when T-Cell count is adequate





Chicken pox - Varicella


MMR – measles, mumps, rubella




Community Interventions





Education and prevention are the best ways to manage AIDS.


Safe sexual practices


Monogamous relationship


Avoidance of substances such as alcohol and drugs that can cloud judgment.



Changes in HIV





Number of infected newborns has dropped due to treatment of HIV infected
mothers.


HIV has become a chronic disease in children


Team approach


Emphasis on community teaching